Author

P. Sandner

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Friebe· 1 shared C. I. Irion· 1 shared C. Mauersberger· 1 shared E. C. Martinez· 1 shared E. S. Buys· 1 shared F. Wunder· 1 shared H. B. Sager· 1 shared H. Schunkert· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPhysiology

A. Friebe

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Loss of soluble guanylyl cyclase in platelets contributes to atherosclerotic plaque formation and vascular inflammation

C. Mauersberger, H. B. Sager, J. Wobst, T. A. Dang, L. Lambrecht, S. Koplev, M. Stroth, N. Bettaga, J. Schlossmann, F. Wunder, A. Friebe, J. L. Bjoerkegren, L. Dietz, P. Sandner, O. Soehnlein, , T. Kessler

This study shows that loss of soluble guanylyl cyclase in platelets promotes atherosclerosis by reducing angiopoietin-1 release and increasing leukocyte adhesion, suggesting sGC stimulation as a potential therapy for coronary artery disease.

bioRxiv · Nov 15, 2021Physiology

Soluble guanylate cyclase stimulation mitigates skeletal and cardiac muscle dysfunction in a mdx model of Duchenne muscular dystrophy

L. Zhang, Y. Xu, K. Yousefi, C. I. Irion, R. A. Alvarez, S. M. Krishnan, J.-P. Stasch, E. C. Martinez, E. S. Buys, P. Sandner, L. A. Shehadeh, J. M. Percival

This study shows that stimulating soluble guanylate cyclase (sGC) with BAY41-8543 reduces skeletal and cardiac muscle dysfunction in mdx mice, suggesting sGC as a therapeutic target for Duchenne muscular dystrophy.

bioRxiv · Feb 14, 2021 · PreprintPhysiology