Author

R. Deo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Research Focus:protein structure prediction· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Khanna· 1 shared A. S. Perelson· 1 shared C. Shi· 1 shared C. Zitzmann· 1 shared D. A. Wohl· 1 shared D. M. Smith· 1 shared E. S. Daar· 1 shared E. Titus· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperImmunology

A. Khanna

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Author profile →

3 papers

Recent·Most cited

Modeling the emergence of viral resistance for SARS-CoV-2 during treatment with an anti-spike monoclonal antibody

T. Phan, C. Zitzmann, K. W. Chew, D. M. Smith, E. S. Daar, D. A. Wohl, J. J. Eron, J. S. Currier, M. D. Hughes, M. C. Choudhary, R. Deo, J. Z. Li, R. M. Ribeiro, R. Ke, A. S. Perelson

This paper models viral resistance emergence in SARS-CoV-2 treated with anti-spike monoclonal antibodies, showing that target cell replenishment mechanisms can explain high viral rebounds.

bioRxiv · Sep 14, 2023Immunology

Tempered IL-2 Signals Program PD-1 Checkpoint Blockade-Responsive Stem-like Exhausted T Cells During Priming

V. Kalia, J. Reed, A. Khanna, R. Toumi, H. Xiao, T. Pulliam, R. Deo, P. Nghiem, S. Sarkar

This study shows that tempered IL-2 signals promote the development of PD-1 checkpoint blockade-responsive stem-like exhausted CD8 T cells during priming, while strong IL-2 signals drive terminal differentiation.

bioRxiv · May 25, 2023 · PreprintImmunology

The structure of a calsequestrin filament reveals mechanisms of familial arrhythmia

E. Titus, F. Deiter, C. Shi, J. Wojciak, M. Scheinman, N. Jura, R. Deo

This paper reports the crystal structure of a cardiac calsequestrin filament and identifies mutations that disrupt filament formation, linking them to arrhythmia.

bioRxiv · Jul 31, 2019Biochemistry