Author

R. Pierce

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cell-cell communication· 1 Approach:deep learning· 1

Frequent Co-Authors

A. Egozi· 1 shared A. Koehne· 1 shared A. Rodriguez· 1 shared B. McCourt· 1 shared C. Leverich· 1 shared D. Shouval· 1 shared E. Choi· 1 shared E. Gordon· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperImmunology

A. Egozi

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Single cell atlas of the neonatal small intestine with necrotizing enterocolitis

A. Egozi, O. Olaloye, L. Werner, T. Silva, B. McCourt, R. Pierce, X. An, F. Wang, K. Chen, J. Pober, D. Shouval, S. Itzkovitz, L. Konnikova

This study uses single-cell and bulk RNA sequencing to identify cellular changes and interactions in necrotizing enterocolitis, highlighting inflammatory macrophages, T cells, and epithelial-mesenchymal-immune interactions.

bioRxiv · Mar 1, 2022 · PreprintImmunology

Tissue-Specific Biochemical Differences Between Chronic Wasting Disease Prions Isolated From Free Ranging, White-Tailed Deer (Odocoileus virginianus)

K. Wagner, R. Pierce, E. Gordon, J. R. Ballard, J. A. Moreno, M. D. Zabel

This study compares biochemical differences between prions isolated from the brain and lymph node of white-tailed deer with chronic wasting disease.

bioRxiv · Jul 19, 2021Pathology

Helicobacter pylori accelerates KRAS-dependent gastric dysplasia

V. P. O'Brien, A. Koehne, J. Dubrulle, A. Rodriguez, C. Leverich, P. Kong, J. Campbell, R. Pierce, J. Goldenring, E. Choi, N. Salama

This study shows that Helicobacter pylori infection accelerates KRAS-driven gastric dysplasia in mice by promoting inflammation, altering gene expression, and increasing proliferation of preneoplastic glands.

bioRxiv · Oct 6, 2020Cancer Biology