Author

S. Apcher

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:proteomics· 3 Research Focus:cancer & tumor microenvironment· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

A. Pierson· 2 shared M. Boulpicante· 2 shared R. Darrigrand· 2 shared A. Bachi· 1 shared A. Cattaneo· 1 shared A. Gueiderikh· 1 shared B. Gaudineau· 1 shared C. Garcia· 1 shared

Activity by Year

2019-2019 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersImmunology

A. Pierson

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Splicing inhibition enhances the antitumor immune response through increased tumor antigen presentation and altered MHC-I immunopeptidome

A. Pierson, R. Darrigrand, M. Rouillon, M. Boulpicante, Z. D. Renko, C. Garcia, M. Ghosh, M.-C. Laiguillon, C. Lobry, M. Alami, S. Apcher

This study shows that splicing inhibition with IP2 enhances antitumor immunity by increasing antigen presentation and altering MHC-I immunopeptidomes.

bioRxiv · Jan 6, 2019 · PreprintImmunology

Tumors escape immunosurveillance by overexpressing the proteasome activator REGγ

M. Boulpicante, R. Darrigrand, A. Pierson, V. Salgues, B. Gaudineau, M. Khaled, A. Cattaneo, A. Bachi, P. Cascio, S. Apcher

The paper shows cancer cells evade immunosurveillance by upregulating REGγ, which increases destruction of MHC-I peptides generated from nuclear translation products.

bioRxiv · Jan 6, 2019Immunology

Fanconi anemia proteins are required to maintain nucleolar homeostasis.

A. Gueiderikh, G. Rouvet, S. Souquere-Besse, S. Apcher, J.-J. Diaz, F. Rosselli

This study shows that Fanconi anemia proteins are essential for maintaining nucleolar homeostasis, with loss-of-function leading to nucleolar DNA damage, rDNA rearrangements, and impaired ribosome biogenesis.

bioRxiv · Jan 2, 2019 · PreprintCell Biology