Author

S. Pozzi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:proteomics· 1 Organism:zebrafish· 1

Frequent Co-Authors

A. A. Ricci· 1 shared A. Dijkstra· 1 shared A. Leikin-Frenkel· 1 shared A. Sacconi· 1 shared A. Schaffer· 1 shared A. Y. Roman· 1 shared D. Merino· 1 shared E. Pokrishevsky· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

A. A. Ricci

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Inactivation of p53 drives breast cancer brain metastasis by altering fatty acid metabolism

K. Laue, S. Pozzi, Y. Cohen-Sharir, T. Winkler, Y. Eliezer, S. Israeli Dangoor, A. Leikin-Frenkel, K. Lange, J. Zerbib, A. A. Ricci, A. Sacconi, J. Berthelet, A. Schaffer, W. Shi, Y. Liao, , U. Ben-David

This study shows that p53 inactivation promotes breast cancer brain metastasis by altering fatty acid metabolism, with implications for therapeutic targeting.

bioRxiv · Dec 20, 2023 · PreprintCancer Biology

Tryptophan residues in TDP-43 and SOD1 mediate the cross-seeding and toxicity of SOD1

E. Pokrishevsky, M. G. Duval, L. McAlary, S. Louadi, S. Pozzi, A. Y. Roman, S. S. Plotkin, A. Dijkstra, J.-P. Julien, W. T. Allison, N. R. Cashman

This study identifies tryptophan residues in SOD1 and TDP-43 that mediate cross-seeding and toxicity in ALS, suggesting a druggable target for therapeutic intervention.

bioRxiv · Jul 27, 2020 · PreprintCell Biology