Author

S. Zola

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Organism:Drosophila· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

M. E. Pasini· 2 shared P. Bellosta· 2 shared A. M. Valenza· 1 shared C. Bonfanti· 1 shared F. Destefanis· 1 shared G. Viero· 1 shared G. Viola· 1 shared I. Signoria· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersPhysiology

M. E. Pasini

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2 papers

Recent·Most cited

PhysiologybioRxivJul 6, 2023

A Novel Drosophila Model to Investigate Adipose Tissue Macrophage Infiltration (ATM) and Obesity highlights the Therapeutic Potential of Attenuating Eiger/TNF Signaling to Ameliorate Insulin Resistance and ATM.

Z. Mirzoyan, A. M. Valenza, S. Zola…·P. Bellosta

This study develops a Drosophila model to investigate adipose tissue macrophage infiltration and obesity, showing that reducing Eiger/TNF signaling improves insulin resistance and reduces immune cell infiltration.

DOI 10.1242/dmm.050388

Drosophilaexperimental assay

𝕏 PostBluesky

Cell BiologybioRxivJul 6, 2021

Reduction of nucleolar NOC1 accumulates pre-rRNAs and induces Xrp1 affecting growth and resulting in cell competition in Drosophila

F. Destefanis, V. Manara, S. Santarelli…·P. Bellosta

This study shows that reducing NOC1 in Drosophila leads to defects in ribosome biogenesis, growth, and cell competition through increased Xrp1 and eiger-JNK pathway activation.

DOI 10.1242/jcs.260110

Drosophilagenetic perturbation / CRISPR

𝕏 PostBluesky