T. O. Bozkurt

This study shows that the fungal effector AVR-Pik binds and stabilizes rice HMA proteins, co-opting their function to suppress immunity, while also triggering immunity in plants with integrated HMA domains in NLRs.

This study introduces PASTEL, a platform for continuous HR cell death monitoring in plants, achieving high accuracy in classification using machine learning.

This study identifies Rab3GAPL as a regulator of plant autophagy and immunity by modulating Rab8a and ATG8 interactions.

This study quantifies how weak electric fields affect Phytophthora palmivora zoospore behavior, showing increased germination and directed movement towards the positive pole.

A helper NLR is mutated to evade inhibition by a parasite effector, restoring disease resistance gene activity.

The paper shows that a key functional motif in the effector PexRD54 of Phytophthora mirabilis degenerated after a host jump, leading to reduced interaction with host ATG8 proteins.

The paper shows that activation of plant NLRs Rx and Bs2 leads to oligomerization of the helper NLR NRC2, suggesting a distinct activation model compared to mammalian paired NLRs.

This study identifies chloroplast-extrahaustorial membrane contact sites that regulate callose deposition during plant immune responses to pathogens.

This study shows chloroplasts accumulate at the pathogen interface during Phytophthora infestans infection, forming stromules, and this process is partially actin-dependent but not light-dependent.

This study shows that the bacterial effector XopL suppresses plant autophagy by degrading SH3P2, revealing a novel pathogen strategy to evade host defense.

This study shows that the NLR protein NRC4 dynamically changes its subcellular localization during plant-pathogen interaction, accumulating at the haustorium interface before activation and redistributing after immune recognition.

This study identifies host processes targeted by Phytophthora infestans RXLR effectors through protein-protein interaction screens, revealing vesicle trafficking as a major effector-targeted process.

This study shows how the potato blight pathogen P. infestans uses the effector PexRD54 to redirect host autophagy towards the pathogen interface, benefiting the pathogen.

This study identifies a large NLR immune signaling network in plants that provides broad immunity to multiple pathogen types.