Author

T. Pourcher

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 3 Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:genetic perturbation / CRISPR· 1 Assay:imaging· 1

Frequent Co-Authors

J. Guglielmi· 2 shared A. Schiazza· 1 shared B. Cambien· 1 shared C. C. Feral· 1 shared C. Delannoy· 1 shared C. Iltis· 1 shared D. Kang· 1 shared E. Cosson· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersPhysiology

J. Guglielmi

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

New insights into iodide metabolism based on preclinical models: impact on radiotherapy efficacy and protection against radioactive iodine exposure.

J. Guglielmi, G. D'Andrea, F. Graslin, K. Chatti, A. Schiazza, S. Lindenthal, J. Darcourt, B. Cambien, T. Pourcher

This study investigates iodine metabolism in mice and rats, showing diet and KI administration effects on thyroid and non-thyroidal organs, and provides insights for radiotherapy and radiation protection.

bioRxiv · Oct 11, 2023 · PreprintPhysiology

Telomere elongation in the gut extends zebrafish lifespan

M. El Mai, J.-M. GUIGONIS, T. POURCHER, D. Kang, J.-X. Yue, M. G. Ferreira

This study shows that gut-specific telomerase activity in zebrafish prevents premature aging and extends lifespan by rescuing gut dysfunction and its systemic effects.

bioRxiv · Jan 10, 2022 · PreprintCell Biology

A ganglioside-based senescence-associated immune checkpoint

C. Iltis, L. Seguin, L. Cervera, L. Duret, T. Hamidouch, S. Kunz, O. Croce, C. Delannoy, Y. Guerardel, F. Allain, L. Moudombi, P. Hofman, E. Cosson, J. Guglielmi, T. Pourcher, , J. Cherfils-Vicini

This paper identifies GD3 as a senescence-associated immune checkpoint that suppresses NK cell-mediated clearance of senescent cells, showing that targeting GD3 can enhance anti-fibrotic effects in a mouse model.

bioRxiv · Apr 23, 2021 · PreprintImmunology