Author

Z. Hegedus

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:fungi (other)· 1 Research Focus:host-pathogen & immune response· 1 Assay:metabolomics· 1 Organism:mouse· 1

Frequent Co-Authors

A. A. Ibarra· 1 shared A. Angyal· 1 shared A. Gacser· 1 shared A. Goodall· 1 shared A. J. Wilson· 1 shared A. Szekeres· 1 shared C. C. Shoulders· 1 shared C. Scazzocchio· 1 shared

Activity by Year

2019-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperBiochemistry

A. A. Ibarra

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Understanding p300-transcription factor interactions using sequence variation and hybridization

F. Hobor, Z. Hegedus, A. A. Ibarra, V. L. Petrovicz, G. J. Bartlett, R. B. Sessions, A. J. Wilson, T. A. Edwards

The paper investigates HIF-1 sequence variation tolerance using biochemical assays, showing that sequence changes are tolerated through disordered binding interactions.

bioRxiv · Dec 16, 2021Biochemistry

Nicotinate degradation in a microbial eukaryote: a novel, complete pathway extant in Aspergillus nidulans

E. Bokor, J. Amon, M. Varga, A. Szekeres, Z. Hegedus, T. Jakusch, M. Flipphi, C. Vagvolgyi, A. Gacser, C. Scazzocchio, Z. Hamari

The paper identifies a complete eukaryotic nicotinate utilization pathway in Aspergillus nidulans, revealing novel metabolites and enzymes distinct from bacterial pathways.

bioRxiv · Aug 17, 2021 · PreprintMicrobiology

Myeloid Tribbles 1 induces early atherosclerosis via enhanced foam cell expansion

J. Johnston, A. Angyal, R. Bauer, S. Hamby, K. Suvarna, K. Baidzajevas, Z. Hegedus, N. Dear, M. Turner, The Cardiogenics Consortium, H. Wilson, A. Goodall, D. J. Rader, C. C. Shoulders, S. Francis, E. Kiss-Toth

This study shows that myeloid Trib1 promotes early atherosclerosis by enhancing foam cell formation through increased lipid uptake and OLR1 expression.

bioRxiv · May 5, 2019Immunology