Author

S. Hamby

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

E. Kiss-Toth· 3 shared A. H. Goodall· 2 shared J. Johnston· 2 shared The Cardiogenics Consortium· 2 shared A. Angyal· 1 shared A. Goodall· 1 shared A. Ocana· 1 shared C. C. Shoulders· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersImmunology

E. Kiss-Toth

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Ageing-related defects in macrophage function are driven by MYC and USF1 transcriptional programmes

C. E. Moss, S. Johnston, M. Clements, V. Codd, S. Hamby, A. H. Goodall, S. Desmukh, I. M. Sudbery, D. Coca, H. L. Wilson, E. Kiss-Toth

This study identifies MYC and USF1 as key drivers of age-related functional decline in human macrophages, showing their role in phagocytosis, migration, and morphology.

bioRxiv · Sep 20, 2023 · PreprintImmunology

TRIB1 regulates tumour growth via controlling tumour-associated macrophage phenotypes and is associated with breast cancer survival and treatment response

T. Kim, J. Johnston, F. J. C. Felipe, S. Hamby, S. Castillo-Lluva, The Cardiogenics Consortium, A. H. Goodall, G. Velasco, A. Ocana, M. Muthana, E. Kiss-Toth

This paper shows TRIB1 in macrophages affects breast cancer growth and treatment response by altering cytokine levels and immune cell composition.

bioRxiv · Jun 7, 2021 · PreprintCancer Biology

Myeloid Tribbles 1 induces early atherosclerosis via enhanced foam cell expansion

J. Johnston, A. Angyal, R. Bauer, S. Hamby, K. Suvarna, K. Baidzajevas, Z. Hegedus, N. Dear, M. Turner, The Cardiogenics Consortium, H. Wilson, A. Goodall, D. J. Rader, C. C. Shoulders, S. Francis, E. Kiss-Toth

This study shows that myeloid Trib1 promotes early atherosclerosis by enhancing foam cell formation through increased lipid uptake and OLR1 expression.

bioRxiv · May 5, 2019Immunology