Rafael Koch

This study shows that reducing NonA in Drosophila worsens TDP-43-induced pathologies, suggesting a functional link between NONO and TDP-43 in proteinopathy.

This study identifies cis-regulatory divergence in the Pdf gene as a driver of circadian behavioral plasticity evolution between Drosophila species.

The paper identifies vulnerable DA neuron subpopulations in Drosophila PD models using split-GAL4 and highlights an age-related caveat of the system.

This study identifies nitric oxide as a critical signaling molecule linking circadian pacemaker neurons to locomotor behavior in Drosophila, with glia as a major source of NO.