Author

Denise Jahn

Charité – Universitätsmedizin BerlinORCID 0000-0001-8912-5851Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:mouse· 2 Assay:behavioral assay· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Adibeh Rahmani· 1 shared Andranik IvanoviD· 1 shared Anke BaranowskyiD· 1 shared Ann-Kathrin Meß· 1 shared Anne Schlutt· 1 shared Antonia DonatiD· 1 shared Bernhard HiebliD· 1 shared Christine GraffmanniD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

Adibeh Rahmani

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

ImmunologyPLoS ONEPeer-reviewedMay 7, 2026

A novel application tunnel in combination with medical training reduces stress induced by frequent intraperitoneal injections and blood sampling in mice

Anne Schlutt, Katarina Riesner, Merle Kochan…·Linda Hammerich

This study introduces a medical training program with an application tunnel to reduce stress in mice during frequent intraperitoneal injections and blood sampling.

DOI 10.1371/journal.pone.0341404

mousebehavioral assay

𝕏 PostBluesky

Cell BiologyScience Translational MedicinePeer-reviewedApr 17, 2024

Increased beta2-adrenergic signaling is a targetable stimulus essential for bone healing by promoting callus neovascularization

Denise Jahn, Paul Richard Knapstein, Ellen Otto…·Johannes Keller

This study identifies beta2-adrenergic signaling as a key driver of bone healing by enhancing callus neovascularization, with implications for therapeutic strategies and the risks of beta-blockers.

42 citationstop 1%

mouseexperimental assay

𝕏 PostBluesky