Author

Maike Herrmann

Paul Ehrlich InstitutORCID 0000-0002-0832-3539Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Maike Herrmann1 paper

Biology Disciplines

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Bioinformatics· 1 Microbiology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Research Focus:differential expression· 3 Organism:human· 3 Approach:statistical model· 3 Research Focus:cell type annotation· 1 Research Focus:database / resource· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

Csaba MiskeyiD· 3 shared Eike SteinmanniD· 2 shared Nicola FrericksiD· 2 shared Yannick BrüggemanniD· 2 shared André GömeriD· 1 shared Bevan SawatskyiD· 1 shared Bingqian QuiD· 1 shared Birthe M. Reinecke· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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3 shared papersMicrobiology

Csaba MiskeyORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

MicrobiologybioRxivPreprintJan 21, 2026

Yellow fever vaccine propagation in primary human hepatocytes triggers antiviral and cytolytic responses

Robin D. V. Kleinert, Maike Herrmann, André Gömer…·Richard J. P. Brown

This study compares antiviral and cytolytic responses in primary human hepatocytes to YFV vaccine and virulent strains, finding similar propagation kinetics but distinct transcriptional responses.

DOI 10.64898/2026.01.21.700765

humanbulk RNA-seq

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BioinformaticsbioRxivPreprintNov 13, 2024

TSniffer: Unbiased de novo identification of RNA editing sites and quantification of editing activity in RNA-seq data

Maike Herrmann, Yvonne Krebs, Francisco M Acosta…·Christian Karl Pfaller

TSniffer identifies RNA editing sites and quantifies editing activity in RNA-seq data, revealing widespread ADAR-mediated editing in mammals.

1 citation

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Molecular BiologybioRxivPreprintNov 22, 2023

Hepatitis C virus cell culture adaptive mutations enhance cell culture propagation by multiple mechanisms but boost antiviral responses in primary human hepatocytes

Nicola Frericks, Richard J.P. Brown, Birthe M. Reinecke…·Julie Sheldon

This study identifies cell culture adaptive mutations in HCV that enhance replicative fitness in cell lines but reduce virion production in primary human hepatocytes by boosting antiviral responses.

DOI 10.1101/2023.11.22.568224

humanbulk RNA-seq

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