Author

Anand Bhushan

Cleveland ClinicORCID 0000-0002-0966-0289Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1

Frequent Co-Authors

Belinda B. WillardiD· 2 shared Connie K. P. TamiD· 2 shared Jonathan ChaniD· 2 shared Yan SuniD· 2 shared Anchal AgarwaliD· 1 shared Anton A. KomariD· 1 shared Daniel J. LindneriD· 1 shared Elise G. Holvey-Bates· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersCell Biology

Belinda B. WillardORCID

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3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintMar 13, 2026

METTL16 promotes taxane resistance in Triple-Negative Breast Cancer through m6A-dependent translational upregulation of ABCB1

Elise G. Holvey-Bates, Jesse A. Coker, Daniel J. Lindner…·Sarmishtha De

This study identifies METTL16 as a driver of taxane resistance in TNBC by enhancing ABCB1 translation via m6A modification, showing its therapeutic potential.

1 citation

humanbulk RNA-seq

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Cell BiologybioRxivPreprintJan 4, 2024

Epithelial cytokeratin 6a restricts secretory autophagy of proinflammatory cytokines by interacting with Sec16A

Anand Bhushan, Jonathan Chan, Yan Sun…·Connie K. P. Tam

This study shows that K6a restricts secretory autophagy of proinflammatory cytokines by interacting with Sec16A, reducing cytokine secretion in corneal epithelial cells.

1 citation

mouseproteomics

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ImmunologybioRxivPreprintOct 24, 2023

Keratin 6a attenuates Toll-like receptor-triggered proinflammatory response in corneal epithelial cells by suppressing ELKS/IKKϵ-dependent activation of NF-κB

Jonathan Chan, Yan Sun, Anand Bhushan…·Connie K. P. Tam

This study shows that keratin 6a suppresses TLR-triggered inflammation in corneal epithelial cells by modulating ELKS/IKKε-dependent NF-κB activation, preventing excessive cytokine and chemokine production.

1 citation

mouseproteomics

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