Radhika P. Atit

This study investigates the role of Fibronectin 1 in suture patency and its dysregulation in craniosynostosis models in mice, identifying FN1 as a key regulator of calvarial growth and cell differentiation.

This study shows that the Wnt/lipogenesis axis mediates lipodystrophy and recovery in skin fibrosis by regulating lipid content and extracellular matrix.

This study shows that Wnt activation in mature dermal adipocytes causes lipodystrophy and skin fibrosis, which can be reversed by stopping Wnt activation.

This study uses automated image analysis to evaluate changes in collagen topography during skin fibrosis and recovery in a Wnt-induced model.

This study identifies the role of mesenchymal Wnts in regulating morphogenetic movements of calvarial osteoblasts during apical expansion in mouse embryos.

This study shows that graded Fibronectin (FN1) cues guide calvarial osteoblast migration and suture patency, with disruptions in FN1 or related pathways causing craniosynostosis in mice.

The paper shows that Wnt and ERK signaling pathways work together to repress Sox9 and control cell fate in cranial mesenchyme during calvarial development.

This study shows dermal EZH2 coordinates dermal fibroblast differentiation and epidermal proliferation during mouse skin development by regulating Wnt/β-catenin and RA signaling.

The paper shows that oscillatory cortical forces drive 3D cell intercalations in the mouse mandibular arch, shaping embryonic structures through Wnt5a, YAP/TAZ, and PIEZO1.