Kazuaki Homma

This study reveals how prestin translates membrane tension into charge and motor movement during sound-evoked vibrations through structural analysis and complementary experiments.

This study investigates how mutations in prestin affect cochlear amplification and hearing, finding that reduced prestin function leads to frequency-specific hearing loss but not complete deafness.

This study identifies common and distinct molecular mechanisms among SLC26 proteins using mutations in SLC26A4, SLC26A5, and SLC26A9.

The paper functionally characterizes deafness-associated pendrin and prestin variants using in vitro assays to assess their pathogenic impacts.