Dagmar Frahm

This study identifies Monalysin, a pore-forming toxin from Pseudomonas entomophila, as a factor that suppresses defecation in infected Drosophila females.

This study shows sex differences in Drosophila intestinal metabolism affect infection outcomes, with males clearing pathogens via antioxidant pathways while females experience higher pathogen virulence.

This study shows that gut microbiota, specifically Lactiplantibacillus plantarum, enhances fatty acid synthesis in Drosophila, promoting pathogen virulence and immune-mediated damage.

This study identifies host genetic variants linked to Lactiplantibacillus plantarum abundance in Drosophila melanogaster and shows heparan sulfate synthesis promotes microbiota expansion and intestinal defense during infection.

This study shows that MprF in Lactiplantibacillus plantarum helps it survive in Drosophila gut during inflammation by reducing immune response and maintaining microbial stability.

This study identifies Spiroplasma poulsonii as enhancing Drosophila melanogaster resistance to pathogens through iron sequestration and melanization.