Igor Iatsenko

This study identifies Monalysin, a pore-forming toxin from Pseudomonas entomophila, as a factor that suppresses defecation in infected Drosophila females.

This study shows sex differences in Drosophila intestinal metabolism affect infection outcomes, with males clearing pathogens via antioxidant pathways while females experience higher pathogen virulence.

This study shows that gut microbiota, specifically Lactiplantibacillus plantarum, enhances fatty acid synthesis in Drosophila, promoting pathogen virulence and immune-mediated damage.

This study shows that sex differences in iron sequestration via Transferrin 1 affect infection outcomes in Drosophila, with males being more resistant to Providencia alcalifaciens.

The paper identifies a novel pacemaker activity in Drosophila midgut called superstalsis, linked to specific muscle activation and gene expression under physiological conditions.

This study identifies host genetic variants linked to Lactiplantibacillus plantarum abundance in Drosophila melanogaster and shows heparan sulfate synthesis promotes microbiota expansion and intestinal defense during infection.

The paper tracks the colonization and evolution of opportunistic pathogens in critically ill patients, identifying a mutation linked to healthcare-associated infections.

This study shows that MprF in Lactiplantibacillus plantarum helps it survive in Drosophila gut during inflammation by reducing immune response and maintaining microbial stability.

This study identifies Spiroplasma poulsonii as enhancing Drosophila melanogaster resistance to pathogens through iron sequestration and melanization.