Author

Min Joo Kim

Gladstone InstitutesORCID 0000-0002-7906-0115Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 4

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 4 Research Focus:cell type annotation· 4 Research Focus:differential expression· 4 Organism:mouse· 4 Approach:experimental assay· 3 Research Focus:gene regulatory network inference· 2 Approach:statistical model· 2 Research Focus:cell-cell communication· 1

Frequent Co-Authors

Jessica R. BlumenfeldiD· 4 shared Oscar YipiD· 4 shared Yadong HuangiD· 4 shared Yanxia Hao· 4 shared Claire Ellis· 3 shared David M. ShostakiD· 3 shared Jeremy Nguyen· 3 shared Kaylie Suan· 3 shared

Activity by Year

2023-2025 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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4 shared papersNeuroscience

Jessica R. BlumenfeldORCID

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4 papers

Recent·Most cited

NeurosciencebioRxivPreprintDec 13, 2025

Neuronal APOE4 reduction with an APOE-I3-targeting ASO protects against neurodegeneration and neuroinflammation in an Alzheimer's disease mouse model

Oscar Yip, Leon Yao, Jessica R. Blumenfeld…·Yadong Huang

DOI 10.64898/2025.12.10.693468

mousebulk RNA-seq

𝕏 PostBluesky

NeurosciencebioRxivPreprintDec 9, 2025

Neuronal APOE4 drives damaging lipid accumulation via contact-dependent neuron-oligodendrocyte-microglia interaction in Alzheimer's disease

Min Joo Kim, Jessica R. Blumenfeld, Yaqiao Li…·Yadong Huang

This study shows neuronal APOE4 drives lipid accumulation in AD through neuron-oligodendrocyte-microglia interactions, with microglia showing preferential lipid buildup.

1 citation

mousebulk RNA-seq

𝕏 PostBluesky

NeurosciencebioRxivPreprintNov 25, 2025

Neuronal APOE4 alone is sufficient to drive tau pathology, neurodegeneration, and neuroinflammation in an Alzheimer's disease mouse model

Jessica R. Blumenfeld, Yaqiao Li, Min Joo Kim…·Yadong Huang

This study shows neuronal APOE4 alone can drive tau pathology, neurodegeneration, and neuroinflammation in a mouse model, similar to a human APOE4 knock-in model.

2 citations

mousebulk RNA-seq

𝕏 PostBluesky

NeurosciencebioRxivPreprintNov 10, 2023

Microglia Depletion Reduces Human Neuronal APOE4-Driven Pathologies in a Chimeric Alzheimer's Disease Model

Antara T. Rao, Nuo Chen, Min Joo Kim…·Yadong Huang

The paper investigates the role of microglia in APOE4-driven Alzheimer's pathology using a chimeric model, finding that microglia are required for APOE4-induced pathologies.

8 citations

𝕏 PostBluesky