Tabea O. C. Moll

This study shows that loss of asgr1a in zebrafish leads to excess dietary cholesterol excretion rather than hepatic accumulation, suggesting a role for ASGR1 in cholesterol metabolism.

The paper develops a zebrafish reporter to measure atherogenic lipoprotein kinetics and finds that lipolysis is suppressed by high-fat meals.

This study identifies molecular mechanisms of resistance to immune checkpoint blockade in metastatic melanoma using proteogenomic and spatial analyses of tumor and immune microenvironment data.

This study shows that a high-cholesterol diet in zebrafish leads to hypercholesterolemia and fasting-induced liver triglyceride accumulation, with lipid accumulation in adipocytes contributing to hepatic steatosis.

The paper introduces a method to raise individual juvenile zebrafish in a recirculating system for experiments requiring repeated measures, showing growth comparable to conventional methods.