Richard D. Cummings

The paper introduces N-glycopedia, a resource of N-glycan standards and analytical methods for structural analysis, enabling more accurate glycomics research.

This study identifies disrupted O-GalNAc glycosylation as a mechanism and biomarker for SLC35A2-associated epilepsy using mouse models and human tissue analysis.

This study identifies protective antibodies against Mycobacterium tuberculosis and shows that their Fc-effector function and Fab domains collaborate to restrict infection through immune cell reorganization.

This paper maps intestinal glycans across development, diet, and inflammation in mice and humans, identifying dynamic changes and microbiome influences.

The paper investigates the N-glycome changes during neuronal differentiation and finds that brain-specific N-glycans like Man-5 are programmed independently of the transcriptome.

The paper identifies O-GalNAc glycans' spatial enrichment in white matter and their role in regulating nodes of Ranvier through glycoproteomic analysis and functional experiments.

The paper identifies distinct spatial distributions of N-glycans in the mouse brain using lectin-based staining.

This study shows that the schizophrenia-associated SLC39A8 variant A391T alters N-glycosylation in the mouse brain, affecting glycoproteins involved in schizophrenia-related pathways.

The paper analyzes glycosylation patterns in mouse brain regions and sexes, finding reduced complexity and minimal sex differences compared to plasma.

This study maps N-glycans on ZIKV E proteins and shows their role in viral entry via host lectins, highlighting their importance in pathogenesis.