Anna Hayes

This study shows that an AGE-rich diet during adolescence impairs memory via hippocampal synaptic deficits and microbiome changes, which can be mitigated by AGE inhibitors.

This study shows that oxytocin neurons in the paraventricular and supraoptic hypothalamic nuclei have opposing effects on food intake, with PVH neurons reducing meal size and SON neurons increasing it.

This study shows that early-life food insecurity in rats leads to memory impairments in adulthood, despite normal metabolic and behavioral outcomes.

This study shows that vagus nerve-mediated acetylcholine release in the dorsal hippocampus supports memory function, and its disruption contributes to obesity-induced cognitive impairments.

This study shows that endotoxemia-induced inflammation without obesity reduces anxiety-like and impulsive behavior but does not impair learning and memory in rats.

This study shows that oxytocin signaling in the dorsal hippocampus promotes prosocial eating in rats by enhancing food consumption with familiar conspecifics and facilitating social transmission of food preference.

This study shows that a western diet during development impairs memory by disrupting hippocampal acetylcholine signaling, which can be partially reversed with pharmacological intervention.

This study shows that early-adolescent Western diet exposure impairs memory in male rats long-term, but not in females, while late-adolescent exposure only affects males temporarily.

This study identifies hypothalamic MCH neurons that integrate food-related appetitive and consummatory processes through physiological and behavioral responses.

This study shows that early-life low-calorie sweetener consumption in rodents leads to long-term metabolic, behavioral, and neural impairments without weight gain.

This study shows that adolescent Western diet consumption in female rats leads to long-lasting memory and gut microbiome issues that are not fully reversed by adult dietary changes.