Rodrigo Fernández‐González

The paper investigates whether tissue-level mechanics during Drosophila germ-band extension influence neuroblast ingression dynamics and finds that cell-intrinsic mechanisms maintain ingression kinetics despite changes in tissue fluidity.

This study identifies Rab4 and Rab11 GTPases as essential for reinforcing adherens junctions at the wound edge through E-cadherin recycling, promoting rapid embryonic wound healing.

The paper shows that cell divisions in the Drosophila embryo challenge and refine tissue boundaries by reducing tension and increasing cell motility.

This study uses simulations and live cell imaging to distinguish active and passive mechanical drivers of convergent extension in developing tissues.

This study shows that mTORC1 inhibits autophagy to enable cell swelling, which accelerates embryonic wound repair.

This study shows that Cofilin promotes F-actin turnover at wound edges in Drosophila embryos, maintaining a flexible actin network to enable rapid wound closure.

ReSCU-Nets improve segmentation of multidimensional microscopy data by using previous frame results as prompts, outperforming existing models in time-lapse sequences.

This study identifies myosin waves and a mechanical asymmetry as key factors in the oscillatory migration of Drosophila cardiac progenitors.

This study identifies the role of the Crumbs complex in regulating apical membrane dynamics during epithelial cell ingression, showing its necessity for proper timing and execution of the process.