Emily J. Illingworth

This study examines gene expression changes in fetal liver, heart, lung, and placenta after prenatal arsenic exposure in mice, identifying immune-related pathway alterations and reduced Gbp3 expression in female placentas.

This study shows that low-dose inorganic arsenic exposure reprograms macrophage polarization, increasing tumor-promoting activity and altering immune responses in a sex-dependent manner.

This study shows that gestational arsenic exposure increases postpartum heart size and disrupts calcium handling in cardiomyocytes.

This study shows that prenatal arsenic exposure reduces maternal cardiac growth and alters gene expression in late pregnancy, potentially contributing to cardiovascular disease.

This study examines how low-dose inorganic arsenic exposure affects gene expression in murine bone marrow-derived macrophages, revealing sex- and stimulation-dependent changes in immune-related pathways.