Małgorzata Tokarska-Schlattner

The paper shows ACSS2 does not generate butyryl- or crotonyl-CoA from SCFAs, challenging previous assumptions about its substrate specificity.

The paper compares methods to quantify acyl-CoA species in rat liver under normal and ischemic conditions, finding ischemia affects short chain acyl-CoA concentrations differently based on duration.

This study shows that NME1/2 regulate liver response to high-fat diet by repressing de novo lipogenesis and activating protective gene expression through histone acetylation.