Author

Xingyuan Liu

Chinese University of Hong KongORCID 0000-0003-4833-0765Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:scRNA-seq· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Assay:DNA methylation· 1

Frequent Co-Authors

Huating WangiD· 3 shared Yang LiiD· 3 shared Chuhan LiiD· 2 shared Hao SuniD· 2 shared Ting XieiD· 2 shared Yulong QiaoiD· 2 shared Bénédicte ChazaudiD· 1 shared Catherine C. L. WongiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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3 shared papersCell Biology

Huating WangORCID

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3 papers

Recent·Most cited

Cell BiologybioRxivPreprintSep 22, 2026

Multiomics Characterization Identifies S1P-secreting USSHigh Skeletal Muscle Stem Cells as Essential Drivers of Niche Remodeling and Muscle Regeneration

Xingyuan Liu, Yingkun Zhang, Yang Li…·Huating Wang

The paper identifies a senescent-like muscle stem cell subpopulation that drives muscle regeneration through secretory activity and cell communication.

DOI 10.64898/2026.09.18.752516

mousescRNA-seq

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Cell BiologyNature CommunicationsPeer-reviewedJul 5, 2025

Multiomics mapping and characterization of cellular senescence in aging human skeletal muscle uncovers a novel senotherapeutic for sarcopenia

Yang Li, Chuhan Li, Qin Zhou…·Huating Wang

This study uses single-nucleus multiomics to characterize senescence in human skeletal muscle, identifies SASP factors, and proposes Maraviroc as a senotherapeutic for sarcopenia.

58 citationstop 1%

humanscRNA-seq

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Cell BiologyNature CommunicationsPeer-reviewedFeb 3, 2025

Skeletal Muscle Stem Cells Modulate Niche Function in Duchenne Muscular Dystrophy through YY1-CCL5 Axis

Yang Li, Chuhan Li, Qiang Sun…·Huating Wang

This study shows that skeletal muscle stem cells modulate their niche in Duchenne muscular dystrophy via the YY1-CCL5 axis, influencing macrophage and fibro-adipogenic progenitor behavior and offering a therapeutic target.

27 citationstop 2%

mousebulk RNA-seq

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