Author

Madison C. McElliott

University of MichiganORCID 0009-0004-2803-3530Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1

Frequent Co-Authors

Abdul A. Soofi· 1 shared Anas Al-Suraimi· 1 shared Asha Claire Telang· 1 shared Edgar A. OttoiD· 1 shared Gregory R. DressleriD· 1 shared Jeffrey A. BeamishiD· 1 shared Jenna T. Ference-Salo· 1 shared Joel M. Weinberg· 1 shared

Activity by Year

2023-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPathology

Abdul A. Soofi

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1 paper

Recent·Most cited

PathologyKidney InternationalPeer-reviewedOct 5, 2023

Pax Protein Depletion in Proximal Tubules Triggers Conserved Mechanisms of Resistance to Acute Ischemic Kidney Injury and Prevents Transition to Chronic Kidney Disease

Jeffrey A. Beamish, Asha Claire Telang, Madison C. McElliott…·Gregory R. Dressler

This study shows that deleting Pax2 and Pax8 in proximal tubules reduces acute kidney injury severity and prevents chronic disease by inducing protective gene expression similar to preconditioning.

17 citations

mousegenetic perturbation / CRISPR

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