Author

Jeffrey A. Beamish

University of MichiganORCID 0000-0003-4851-3481Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1 Organism:rat· 1

Frequent Co-Authors

Jenna T. Ference-Salo· 2 shared Abdul A. Soofi· 1 shared Aisaku NakamuraiD· 1 shared Anas Al-Suraimi· 1 shared Asha Claire Telang· 1 shared Avery Volz· 1 shared Bhagwat PrasadiD· 1 shared Brandi C Bull· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersDevelopmental Biology

Jenna T. Ference-Salo

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2 papers

Recent·Most cited

Developmental BiologybioRxivPreprintSep 3, 2026

Megalin (LRP2), prenatal betamethasone, and injury susceptibility in the developing kidney

Cimran Nakum, Brandi C Bull, Sunitha Yarlagadda…·Meredith Posner Schuh

This study evaluates how prenatal betamethasone exposure and tubular maturation affect gentamicin-induced kidney injury and nephron number in rats.

DOI 10.64898/2026.06.02.729064

ratexperimental assay

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PathologyKidney InternationalPeer-reviewedOct 5, 2023

Pax Protein Depletion in Proximal Tubules Triggers Conserved Mechanisms of Resistance to Acute Ischemic Kidney Injury and Prevents Transition to Chronic Kidney Disease

Jeffrey A. Beamish, Asha Claire Telang, Madison C. McElliott…·Gregory R. Dressler

This study shows that deleting Pax2 and Pax8 in proximal tubules reduces acute kidney injury severity and prevents chronic disease by inducing protective gene expression similar to preconditioning.

17 citations

mousegenetic perturbation / CRISPR

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