Author

A. Amer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Zoology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Assay:behavioral assay· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

A. Badr· 1 shared A. Eddy· 1 shared A. Forero· 1 shared A. Kenney· 1 shared A. Schill· 1 shared A. Tedeschi· 1 shared A. Webb· 1 shared A. Zani· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperImmunology

A. Badr

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The Septin Cytoskeleton is Required for Plasma Membrane Repair

M. I. A. Prislusky, J. G. Lam, V. Ruiz Contreras, M. Ng, M. Chamberlain, S. Pathak-Sharma, M. Fields, X. Zhang, A. Amer, S. M. Seveau

This study identifies SEPT7 as a critical component of the septin cytoskeleton required for plasma membrane repair, showing its role in forming repair domains with F-actin and ANXA2.

bioRxiv · Jul 12, 2023Cell Biology

Physiological phenotypes differ among color morphs in introduced populations of the common wall lizard, Podarcis muralis

A. Amer, S. Spears, P. L. Vaughn, C. Colwell, E. H. Livingston, W. McQueen, A. Schill, D. Reichard, E. J. Gangloff, K. M. Brock

The study examines physiological differences among color morphs of Podarcis muralis in urban environments, finding distinct multivariate phenotypes linked to corticosterone levels.

bioRxiv · Jun 17, 2023 · PreprintZoology

Caspase-4/11 exacerbates disease severity in SARS-CoV-2 infection by promoting inflammation and thrombosis

M. Eltobgy, A. Zani, A. Kenney, S. Estfanous, E. Kim, A. Badr, C. Carafice, K. Daily, O. Whitham, M. Pietrzak, A. Webb, J. Kawahara, A. Eddy, P. Denz, M. Lu, , A. Amer

This study shows that caspase-4/11 worsens SARS-CoV-2 infection by increasing inflammation and thrombosis, independent of GSDMD, suggesting it as a potential drug target for severe COVID-19.

bioRxiv · Sep 24, 2021Immunology