Author

A. Diefenbach

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Research Focus:cell type annotation· 2 Organism:human· 2 Organism:mouse· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Assay:clinical cohort / trial· 1

Frequent Co-Authors

G. A. Heinz· 2 shared M.-F. Mashreghi· 2 shared A. Fagundes· 1 shared A. Kruglov· 1 shared A. Ohnemus· 1 shared A. Rabes· 1 shared A. Radbruch· 1 shared A. Wack· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersImmunology

G. A. Heinz

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

IL-33 controls IL-22-dependent antibacterial defense by modulating the microbiota

I. Roewekamp, L. Maschirow, A. Rabes, F. Fiocca Vernengo, G. A. Heinz, M.-F. Mashreghi, S. Caesar, M. Milek, A. Fagundes, S.-M. Wienhold, G. Nouailles, L. Yao, D. Bruder, J. Boehme, M. Puzianowska-Kuznicka, , B. Opitz

This study shows IL-33 reduces IL-22 production in innate lymphoid cells, increasing resistance to Streptococcus pneumoniae infection, and links genetic variation in IL-33 signaling to human pneumonia.

bioRxiv · Jul 19, 2023Immunology

Induction of cross-reactive antibody responses against the RBD domain of the spike protein of SARS-CoV-2 by commensal microbiota

J. Ninnemann, L. Budzinski, M. Bondareva, M. Witkowski, S. Angermair, J. Kreye, P. Durek, M. Reincke, E. Sanchez-Sendin, S. Yilmaz, T. Sempert, G. A. Heinz, C. Tizian, M. Raftery, G. Schoenrich, , A. Kruglov

This study identifies commensal bacteria that induce cross-reactive anti-RBD IgA antibodies against SARS-CoV-2 in humans and mice.

bioRxiv · Aug 8, 2021 · PreprintMicrobiology

Rotavirus susceptibility of antibiotic-treated mice ascribed to diminished expression of interleukin-22

D. Schnepf, P. Hernandez, T. Mahlakoiv, S. Crotta, M. E. Sullender, S. T. Peterson, A. Ohnemus, C. Michiels, I. Gentle, L. Dumoutier, C. A. Reis, A. Diefenbach, A. Wack, M. T. Baldridge, P. Staeheli

This study shows that antibiotic-treated mice are more susceptible to rotavirus due to reduced IL-22 expression, which normally inhibits viral replication through mechanisms independent of IFN signaling.

bioRxiv · Feb 18, 2021Immunology