Author

A. Jaber

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1 Plant Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Organism:Arabidopsis· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

A. Bigot· 1 shared A. Font-Tello· 1 shared A. K. Tewari· 1 shared A. Toker· 1 shared A. Vu Hong· 1 shared D. Israeli· 1 shared G. Brochier· 1 shared H. Liu· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperBioengineering

A. Bigot

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Disease exacerbation by fibroblast inclusion in Duchenne Muscular Dystrophy MYOrganoids reveals limitations of microdystrophin therapeutic efficacy

L. Palmieri, L. Pili, A. Jaber, A. Vu Hong, M. Marcello, R. El Khoury, G. Brochier, A. Bigot, D. Israeli, I. Richard, S. Albini

This paper develops a human DMD model using MYOrganoids to study fibroblast inclusion effects and evaluates microdystrophin therapy efficacy, finding limited success in reducing fibrosis and improving membrane stability.

bioRxiv · Jul 26, 2023 · PreprintBioengineering

FGFR inhibitor mediated dismissal of SWI/SNF complexes from YAP-dependent enhancers induces adaptive therapeutic resistance

Y. Li, X. Qiu, X. Wang, H. Liu, R. Geck, A. K. Tewari, T. Xiao, A. Font-Tello, K. Lim, J. L. Kristen, M. Morrow, R. Vadhi, P.-L. Kao, A. Jaber, S. Yerrum, , M. Brown

The paper shows that FGFR inhibitors cause SWI/SNF complex dismissal from YAP-dependent enhancers, leading to adaptive resistance in TNBC through epigenetic and metabolic changes.

bioRxiv · Mar 2, 2021Cancer Biology

Expression levels of long noncoding natural antisense transcripts overlapping the UGT73C6 gene affect rosette size of Arabidopsis thaliana

S. K. Meena, M. Heidecker, S. Engelmann, A. Jaber, T. de Vries, K. Baumann-Kaschig, S. Abel, S.-E. Behrens, S. Gago-Zachert

This study identifies two lncNATs overlapping UGT73C6 in Arabidopsis thaliana that regulate rosette size by modulating cell proliferation and leaf growth through transcription factor regulation.

bioRxiv · Mar 1, 2021 · PreprintPlant Biology