Author

A. M. Unni

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Approach:experimental assay· 1 Assay:whole-exome sequencing· 1

Frequent Co-Authors

D. A. Farnsworth· 1 shared D. Lu· 1 shared F. D. Johnson· 1 shared G. de Rappard-Yuswack· 1 shared H. Varmus· 1 shared M. Ladanyi· 1 shared R. Shi· 1 shared R. Somwar· 1 shared

Activity by Year

2016-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCancer Biology

D. A. Farnsworth

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintApr 29, 2022

MEK inhibitor resistance in lung cancer cells associated with addiction to sustained ERK suppression

D. A. Farnsworth, Y. Inoue, F. D. Johnson…·W. Lockwood

The paper identifies drug addiction to MEK inhibitors in KRAS-mutant lung cancer cells, showing that resistance is linked to ERK suppression and KRAS amplification, suggesting therapeutic strategies involving drug holidays.

DOI 10.1101/2022.04.29.490009

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivDec 18, 2016

How Cancer Genomics Drives Cancer Biology: Does Synthetic Lethality Explain Mutually Exclusive Oncogenic Mutations?

H. Varmus, A. M. Unni, W. W. Lockwood

The paper investigates mutual exclusivity of KRAS and EGFR mutations in lung adenocarcinomas, suggesting synthetic lethality explains their absence in co-occurring tumors.

DOI 10.1101/sqb.2016.81.030866

humanwhole-exome sequencing

𝕏 PostBluesky