Author

B. A. McCormick

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

B. M. Mortzfeld· 2 shared S. E. Foley· 2 shared S. K. Bhattarai· 2 shared V. Bucci· 2 shared A. L. Zeamer· 1 shared A. Reboldi· 1 shared C. Bang· 1 shared D. V. Ward· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersImmunology

B. M. Mortzfeld

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Microbiota encoded fatty-acid metabolism expands tuft cells to protect tissues homeostasis during Clostridioides difficile infection in the large intestine

T. D. Kellogg, S. Ceglia, B. M. Mortzfeld, A. L. Zeamer, S. E. Foley, D. Ward, S. K. Bhattarai, B. A. McCormick, A. Reboldi, V. Bucci

This study shows that succinate from the microbiota expands tuft cells in the colon, enhancing type 2 immunity and reducing C. difficile-induced disease.

bioRxiv · Jan 29, 2024 · PreprintImmunology

Microcin MccI47 selectively inhibits enteric bacteria and reduces carbapenem-resistant Klebsiella pneumoniae colonization in vivo when administered via an engineered live biotherapeutic

B. M. Mortzfeld, J. D. Palmer, S. K. Bhattarai, H. L. Dupre, R. Mercado-Lubo, M. W. Silby, C. Bang, B. A. McCormick, V. Bucci

This paper demonstrates that engineered live biotherapeutic Escherichia coli producing microcin MccI47 reduces carbapenem-resistant Klebsiella pneumoniae colonization in mice without disrupting the microbiota.

Gut Microbes · Sep 29, 2022Peer-reviewedBioengineering

The epithelial-specific ER stress sensor IRE1β enables host-microbiota crosstalk to affect colon goblet cell development

M. J. Grey, H. De Luca, D. V. Ward, I. A. M. Kreulen, S. E. Foley, J. R. Thiagarajah, B. A. McCormick, J. R. Turner, W. I. Lencer

This study shows IRE1β is essential for microbiota-induced goblet cell maturation and mucus barrier assembly in the colon, shaping gut microbiota composition through ER stress regulation.

bioRxiv · Jul 28, 2021 · PreprintCell Biology