Author

B. Abraham

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:behavioral assay· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Aviles· 1 shared A. C. Keene· 1 shared A. Clarke· 1 shared A. Durbin· 1 shared A. K B· 1 shared A. K. R· 1 shared A. Madhavan· 1 shared A. Paz· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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1 shared paperEvolutionary Biology

A. Aviles

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Changes in local interaction rules during ontogeny underlie the evolution of collective behavior

A. Paz, K. J. Holt, A. Clarke, A. Aviles, B. Abraham, A. C. Keene, E. R. Duboue, Y. Fily, J. E. Kowalko

The paper studies how local interaction rules change during development and evolution in Mexican tetra fish, showing surface fish develop schooling behavior through alignment and attraction, while cavefish lose schooling behavior.

bioRxiv · Mar 28, 2023Evolutionary Biology

Genetic Predisposition to Neuroblastoma Results from a Regulatory Polymorphism that Promotes the Adrenergic Cell State

N. Weichert-Leahey, H. Shi, T. Tao, D. Oldridge, A. Durbin, B. Abraham, M. Zimmerman, S. Zhu, A. Wood, D. Reyon, J. K. Joung, R. Young, S. J. Diskin, J. Maris, A. T. Look

The paper identifies a regulatory polymorphism in LMO1 that reduces neuroblastoma risk by preventing the adrenergic cell state in zebrafish and humans.

bioRxiv · Feb 28, 2023Cancer Biology

Acetylation of isoniazid - a novel mechanism of isoniazid resistance in Mycobacterium tuberculosis

A. K B, A. Madhavan, B. Abraham, B. M, S. K C, N. P, A. K. R

This study identifies Rv2170 as an acetyltransferase that inactivates isoniazid by acetylation, providing a novel mechanism for isoniazid resistance in Mycobacterium tuberculosis.

bioRxiv · Feb 10, 2020Biochemistry