Author

B. Bone

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

A. Kipar· 1 shared A. Mann· 1 shared A. Zhekova· 1 shared J. L. Coombes· 1 shared J. P. Stewart· 1 shared M. Jefferson· 1 shared P. P. Powell· 1 shared P. Sharma· 1 shared

Activity by Year

2020-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperMicrobiology

A. Kipar

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1 paper

Recent·Most cited

The WD and linker domains of ATG16L1 required for non canonical autophagy limit lethal influenza A virus infection at epithelial surfaces.

Y. Wang, W. Zhang, M. Jefferson, P. Sharma, B. Bone, A. Kipar, J. L. Coombes, T. Pearson, A. Mann, A. Zhekova, Y. Bao, R. A. Tripp, Y. Yamauchi, S. R. Carding, U. Mayer, , T. Wileman

This study shows that non-canonical autophagy, mediated by the WD and linker domains of ATG16L1, protects against lethal influenza A virus infection by limiting viral replication and inflammation in airway epithelial cells.

bioRxiv · Jan 15, 2020 · PreprintMicrobiology