Author

B. C. Schaefer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:imaging· 1 Approach:microscopy & imaging analysis· 1 Organism:mouse· 1

Frequent Co-Authors

A. A. DeLaney· 1 shared B. Demarco· 1 shared B. L. Lee· 1 shared D. Grubaugh· 1 shared D. P. Beiting· 1 shared D. Tovar· 1 shared E. Bjanes· 1 shared E. Krespan· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperImmunology

A. A. DeLaney

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2 papers

Recent·Most cited

Signaling through polymerization and degradation: Analysis and simulations of T cell activation mediated by Bcl10

L. Campanello, M. K. Traver, H. Shroff, B. C. Schaefer, W. Losert

This paper quantifies Bcl10 filament dynamics in T cell activation using microscopy and simulations, showing degradation at filament ends controls response timing.

PLOS Computational Biology · May 20, 2021Peer-reviewedBiophysics

Genetic targeting of Card19 is linked to disrupted Ninj1 expression, impaired cell lysis, and increased susceptibility to Yersinia infection

E. Bjanes, R. G. Sillas, R. Matsuda, B. Demarco, T. Fettrelet, A. A. DeLaney, O. S. Kornfeld, B. L. Lee, E. M. Rodriguez Lopez, D. Grubaugh, M. A. Wynosky-Dolfi, N. H. Philip, E. Krespan, D. Tovar, L. Joannas, , I. E. Brodsky

This study shows that genetic targeting of Card19 disrupts Ninj1 expression, impairs macrophage cell lysis, and increases susceptibility to Yersinia infection.

bioRxiv · Mar 19, 2021 · PreprintImmunology