Author

B. McEwan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Approach:deep learning· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Research Focus:protein folding & dynamics· 1 Assay:protein structure· 1

Frequent Co-Authors

A. Kettenbach· 2 shared B. Lopez-Mendez· 2 shared J. Nilsson· 2 shared M. B. Weisser· 2 shared A. A. Jeyaprakash· 1 shared C. Chan· 1 shared D. Garvanska· 1 shared E. P. Hertz· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersCell Biology

A. Kettenbach

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2 papers

Recent·Most cited

Substrate recognition principles for the PP2A-B55 protein phosphatase

T. Kruse, D. Garvanska, J. K. Varga, W. Garland, B. McEwan, J. Hein, M. B. Weisser, I. B. Puy, C. Chan, P. S. Parrila, B. Lopez-Mendez, A. A. Jeyaprakash, O. Schueler-Furman, T. H. Jensen, A. Kettenbach, J. Nilsson

The paper identifies conserved mechanisms by which PP2A-B55 substrates bind B55 through α-helices and develops a competitive inhibitor to study its function.

bioRxiv · Feb 10, 2024Cell Biology

Cellular toxicity of iHAP1 and DT-061 does not occur through PP2A-B56 targeting

G. Vit, J. Duro, G. Rajendraprasad, E. P. Hertz, L. Holland, M. B. Weisser, B. McEwan, B. Lopez-Mendez, G. Montoya, N. Mailand, K. Maeda, A. Kettenbach, J. Nilsson

This study challenges previous claims that iHAP1 and DT-061 target PP2A-B56 complexes, showing instead that they exert toxicity through microtubule disruption and ER/Golgi disruption, respectively.

bioRxiv · Jul 8, 2021Cell Biology