Author

C. B. M. Porter

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Approach:deep learning· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

A. Regev· 2 shared S. J. Riesenfeld· 2 shared A. E. Neumann· 1 shared A. Jarret· 1 shared A. Subramanian· 1 shared C. A. E. Desel· 1 shared D. A. Dionne· 1 shared D. Dionne· 1 shared

Activity by Year

2018-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersImmunology

A. Regev

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2 papers

Recent·Most cited

ImmunologybioRxivMar 26, 2021

Parental-fetal interplay of immune genes leads to intrauterine growth restriction

G. Kaur, C. B. M. Porter, O. Ashenberg…·L. Fugger

This study identifies a maternal-fetal immune interaction involving KIR2DL1 and HLA-C*05 that causes intrauterine growth restriction in a humanised mouse model.

DOI 10.1038/s41467-022-32171-w

mousespatial transcriptomics

𝕏 PostBluesky

ImmunologybioRxivNov 12, 2018

Skin inflammation driven by differentiation of quiescent tissue-resident ILCs into a spectrum of pathogenic effectors

P. Bielecki, S. J. Riesenfeld, M. S. Kowalczyk…·R. A. Flavell

The paper shows that psoriasis is driven by the reprogramming of quiescent skin-resident ILCs into pathogenic effectors through epigenetic and transcriptional changes.

DOI 10.1038/s41586-021-03188-w

mousescRNA-seq

𝕏 PostBluesky