Author

C. Moro

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Physiology· 2 Pathology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 5 Approach:experimental assay· 4 Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Research Focus:gene regulatory network inference· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:metabolism & bioenergetics· 2

Frequent Co-Authors

V. Bourlier· 3 shared C. Laurens· 2 shared G. Tavernier· 2 shared N. Viguerie· 2 shared A. Astrup· 1 shared A. Batut· 1 shared A. Blais· 1 shared A. Carriere· 1 shared

Activity by Year

2019-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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3 shared papersPhysiology

V. Bourlier

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

ALTERATIONS IN PEROXISOMAL-MITOCHONDRIAL INTERPLAY IN SKELETAL MUSCLE ACCELERATES MUSCLE DYSFUNCTION

M. Scalabrin, E. Turco, L. Nogara, G. Gherardi, G. Trani, S. Negro, A. Franco Romero, Y. Jaspers, E. Baschiera, R. De Cegli, E. Del Prete, T. Cali, B. Blaauw, L. Salviati, M. Rigoni, , V. Romanello

This study shows that disrupting peroxisomal-mitochondrial interactions in skeletal muscle accelerates muscle dysfunction and age-related degeneration.

bioRxiv · Apr 25, 2024Cell Biology

Glucose intolerance in aging is mediated by the Gpcpd1-GPC metabolic axis

D. Cikes, M. Leutner, S. J. - F. Cronin, M. Novatchkova, L. Pfleger, R. Klepochova, E. Rullman, T. Gustafsson, A. Hagelkrueys, C. Knauf, C. Moro, M. Krebs, A. Kautzky-Willer, M. Krssak, M. Orthofer, , N. Viguerie

This study identifies Gpcpd1-GPC metabolic pathway perturbation in aging muscle as a driver of glucose intolerance.

bioRxiv · Oct 26, 2021 · PreprintPhysiology

Nuclear HMGB1 protects from non-alcoholic fatty liver diseases through negative regulation of liver X receptor

J. Personnaz, E. Piccolo, A. Dortignac, J. S. Iacovoni, J. Mariette, A. Polizzi, A. Batut, S. Deleruyelle, R. Paccoud, E. Moreau, F. Martins, T. Clouaire, F. Benhamed, A. Montagner, W. A. Wahli, , J.-P. Pradere

This study shows that nuclear HMGB1 suppresses liver lipogenesis in NAFLD by inhibiting LXR activity, preventing steatosis and insulin resistance.

bioRxiv · Feb 17, 2021 · PreprintPathology

GREM1 is epigenetically reprogrammed in muscle cells after exercise training and controls myogenesis and metabolism

O. Fabre, L. Giordani, A. Parisi, P. Pattamaprapanont, D. Ahwazi, C. E. Brun, I. Chakroun, A. Taleb, A. Blais, E. Andersen, L. R. Ingerslev, P. Maire, M. A. Rudnicki, C. Laurens, K. Citirikkaya, , R. Barres

This study identifies an epigenetic reprogramming of GREM1 in muscle cells after exercise training, which enhances myogenesis and metabolism through the AMPK pathway.

bioRxiv · Feb 20, 2020 · PreprintCell Biology

Atrial natriuretic peptide orchestrates a coordinated physiological response to fuel non shivering thermogenesis

D. Carper, M. Coue, E. B. Nascimento, V. Barquissau, D. Lagarde, C. Pestourie, C. Laurens, J. Vily-Petit, M. Soty, L. Monbrun, M.-A. Marques, Y. Jeanson, Y. Sainte-Marie, A. Mairal, S. Dejean, , C. Moro

This study shows that atrial natriuretic peptide (ANP) promotes cold-induced thermogenesis by enhancing lipolysis and mitochondrial activity in adipocytes, and impairing fuel availability in ANP-deficient mice.

bioRxiv · Dec 6, 2019Physiology