Author

E. J. Brown

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Evolutionary Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

A. Easton· 1 shared A. G. Clark· 1 shared A. M. Whiteley· 1 shared A. Weinstock· 1 shared B. Scolaro· 1 shared C. A. Nikain· 1 shared C. Donahoe· 1 shared C. Hannemann· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperCell Biology

A. Easton

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

SHORT-TERM CALORIC RESTRICTION IN MICE PROMOTES RESOLUTION OF ATHEROSCLEROSIS, WHILE WEIGHT REGAIN ACCELERATES ITS PROGRESSION

B. Scolaro, E. J. Brown, F. Krautter, M. Petitjean, C. Donahoe, S. Pena, M. L. Garabedian, C. A. Nikain, M. Laskou, O. Tufanli, C. Hannemann, M. Aouadi, A. Weinstock, E. Fisher

This study shows that short-term caloric restriction in mice reduces atherosclerosis by promoting Fcgr4+ macrophages, while weight regain accelerates plaque progression by eliminating these cells and reprogramming immune progenitors.

bioRxiv · May 7, 2023 · PreprintImmunology

Copy number evolution in simple and complex tandem repeats across the C57BL/6 and C57BL/10 inbred mouse lines

J. M. Flynn, E. J. Brown, A. G. Clark

The paper estimates copy number change rates in tandem repeats across mouse lines over generations, finding stability in CAG repeats and variation in other repeats.

bioRxiv · Mar 1, 2021Evolutionary Biology

Global proteomics of Ubqln2-based murine models of ALS

A. M. Whiteley, M. A. Prado, S. A. H. de Poot, J. A. Paulo, M. Ashton, S. Dominguez, M. Weber, H. Ngu, J. Szpyt, M. P. Jedrychowski, A. Easton, S. P. Gygi, T. Kurz, M. J. Monteiro, E. J. Brown, D. Finley

The paper identifies proteomic changes in Ubqln2 mutant mice and finds that TRIM32, PEG10, and CXX1B are linked to UBQLN2 function and degradation.

bioRxiv · Feb 22, 2020Cell Biology