Author

E. L. Houlder

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 1

Frequent Co-Authors

A. S. MacDonald· 3 shared J. E. Allen· 2 shared J. E. Konkel· 2 shared L. Boon· 2 shared P. C. Cook· 2 shared A. Chenery· 1 shared A. Morrison· 1 shared B. Dickie· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersImmunology

A. S. MacDonald

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Aspergillus-mediated allergic airway inflammation is triggered by dendritic cell recognition of a defined spore morphotype, a process that can be targeted via antifungal therapeutics.

E. L. Houlder, S. Gago, G. Vere, D. Conn, S. Khan, D. Thomson, M. Shepherd, R. Lebedinec, G. D. Brown, M. Bromley, A. S. MacDonald, P. C. Cook

This study identifies a specific stage of Aspergillus fumigatus spore development that activates dendritic cells to trigger allergic airway inflammation and shows antifungal drugs can reduce this process.

Journal of Allergy and Clinical Immunology · Mar 1, 2025Peer-reviewedImmunology

Mgl2+ cDC2s coordinate fungal allergic airway type 2, but not type 17, inflammation.

P. C. Cook, S. L. Brown, E. L. Houlder, S. Baker, F. R. Svedberg, G. Howell, M. Bertuzzi, L. Boon, J. E. Konkel, J. E. Allen, A. S. MacDonald

The paper identifies Mgl2+ cDC2s as key drivers of type 2, but not type 17, fungal allergic airway inflammation in mice.

bioRxiv · Nov 24, 2023Immunology

Genotype and Th2 cells control monocyte to tissue resident macrophage differentiation during nematode infection of the pleural cavity

C. M. Finlay, J. Parkinson, B. H. Chan, J. Ajendra, A. Chenery, A. Morrison, E. L. Houlder, S. M. Baker, B. Dickie, L. Boon, A. S. MacDonald, J. E. Konkel, D. Rückerl, J. E. Allen

This study shows that host genetics and Th2 cells influence monocyte-to-tissue-resident-macrophage differentiation during nematode infection in mice.

bioRxiv · Dec 17, 2021 · PreprintImmunology