Author

E. Rybalka

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 3 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:cell type annotation· 3 Research Focus:development & morphogenesis· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

C. A. Timpani· 4 shared S. Kourakis· 3 shared D. Debrincat· 2 shared D. Fischer· 2 shared D. G. Campelj· 2 shared G. Spiesberger· 2 shared H. Zhang· 2 shared J. B. de Haan· 2 shared

Activity by Year

2022-2024 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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4 shared papersPathology

C. A. Timpani

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Haptoglobin and glutamine synthetase may biomark cachexia induced by anti-acute myeloid leukemia chemotherapy

D. G. Campelj, C. A. Timpani, G. Spiesberger, L. E. Formosa, J. Steele, H. Zhang, R. B. Schittenhelm, L. Leow, C. Goodman, E. Rybalka

This study shows that the AML chemotherapy regimen induces cachexia in mice, characterized by body and lean mass loss, and identifies haptoglobin as a potential biomarker for chemotherapy-induced cachexia.

bioRxiv · Jul 24, 2024 · PreprintPhysiology

Moderate-term dimethyl fumarate treatment reduces pathology of dystrophic skeletal and cardiac muscle in a mouse model

S. Kourakis, C. A. Timpani, R. M. Bagaric, B. Qi, B. A. Ali, R. Boyer, G. Spiesberger, N. Kandhari, A. L. Peterson, D. Debrincat, T. J. Yates, X. Yan, J. Kuang, J. B. de Haan, N. Stupka, , E. Rybalka

This study shows that moderate-term dimethyl fumarate treatment reduces dystrophic muscle pathology in a mouse model of Duchenne muscular dystrophy, with anti-inflammatory, anti-fibrotic, and anti-lipogenic effects.

bioRxiv · Jul 13, 2024 · PreprintPathology

Loss of endogenous estrogen alters mitochondrial metabolism and muscle clock-related protein Rbm20 in female mdx mice

C. A. Timpani, D. Debrincat, S. Kourakis, R. Boyer, L. E. Formosa, J. R. Steele, H. Zhang, R. B. Schittenhelm, A. P. Russell, E. Rybalka, A. Lindsay

This study shows that estrogen loss in female mdx mice reduces exercise capacity and mitochondrial metabolism, but estrogen replacement therapy can reverse these effects and upregulate Rbm20.

bioRxiv · Feb 8, 2024 · PreprintPathology

Dimethyl fumarate modulates the Duchenne muscular dystrophy disease program following short-term treatment in mdx mice

C. A. Timpani, S. Kourakis, D. A. Debruin, D. G. Campelj, N. Pompeani, N. Dargahi, A. P. Bautista, R. M. Bagaric, E. J. Ritenis, L. Sahakian, P. Hafner, P. G. Arthur, J. R. Terrill, V. Apostolopoulos, J. B. de Haan, , E. Rybalka

This study compares dimethyl fumarate (DMF) and prednisone (PRED) in mdx mice, finding DMF improves muscle function and mitochondrial health while PRED causes side effects.

bioRxiv · Sep 15, 2022 · PreprintPathology