Author

G. Barton

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:cell type annotation· 3 Research Focus:host-pathogen & immune response· 3 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

B. Liu· 2 shared O. Majer· 2 shared A. Marshak-Rothstein· 1 shared A. Reboldi· 1 shared A. Zilinskas· 1 shared B. Hill· 1 shared B. Luckie· 1 shared B. Woo· 1 shared

Activity by Year

2018-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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2 shared papersImmunology

B. Liu

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Kruppel-like Factor (KLF) family members control expression of genes required for serous cavity and alveolar macrophage identities

K. Pestal, L. Slayden, G. Barton

The paper identifies KLF2 and KLF4 as regulators of serous cavity and alveolar macrophage identities through direct gene regulation.

bioRxiv · Feb 28, 2024Immunology

Antigen receptor signaling and cell death resistance controls intestinal humoral response zonation.

F. Raso, S. Liu, M. J. Willett, G. Barton, C. T. Mayer, O. Bannard, M. Acharya, J. Muppidi, A. Marshak-Rothstein, A. Reboldi

This study shows that IgA BCR signaling enhances B cell fitness and promotes diverse IgA responses to gut commensals by rescuing low-affinity B cells and redirecting plasma cell homing.

bioRxiv · Mar 16, 2023Immunology

Cas9+ conditionally immortalized neutrophil progenitors as a tool for genome wide CRISPR screening for neutrophil differentiation and function

R. M. Jong, K. Ching, N. E. Garelis, A. Zilinskas, X. Nguyenla, S. Rawal, B. Hill, B. Luckie, L. Shallow, J. S. Cox, G. Barton, S. Stanley

This paper introduces a Cas9+ER-Hoxb8 neutrophil progenitor cell line for CRISPR screening to study neutrophil differentiation and function, identifying key genes like Cebpe and the WASH complex.

bioRxiv · Jul 19, 2022Immunology

An essential checkpoint for TLR9 signaling is release from 1 Unc93b1 in endosomes

O. Majer, B. Woo, B. Liu, E. Van Dis, G. Barton

The paper identifies a mechanism by which TLR9 is restricted to endosomes through its association with Unc93b1, ensuring proper signaling and preventing autoimmunity.

bioRxiv · Sep 5, 2018 · PreprintImmunology

Unc93b1 recruits Syntenin-1 to dampen TLR7 signaling and prevent autoimmunity

O. Majer, B. Liu, N. Krogan, G. Barton

This study identifies a mechanism by which Unc93b1 recruits Syntenin-1 to specifically inhibit TLR7 signaling and prevent autoimmunity.

bioRxiv · Sep 5, 2018Immunology