Author

G. Gilmer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Approach:statistical model· 2 Approach:diffusion model· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1 Approach:simulation· 1

Frequent Co-Authors

F. Ambrosio· 2 shared H. Iijima· 2 shared A. C. Bean· 1 shared A. D'Amore· 1 shared A. Miller· 1 shared A. Sahu· 1 shared C. Evans· 1 shared D. A. Vorp· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersPathology

F. Ambrosio

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2 papers

Recent·Most cited

A network medicine approach to elucidate mechanisms underlying menopause-induced knee osteoarthritis

G. Gilmer, H. Iijima, N. Jackson, Z. Hettinger, A. C. Bean, J. Bergmann, C. Evans, R. C. Thurston, F. Ambrosio

This study identifies menopause-induced cartilage degradation, senescence, and extracellular matrix disassembly, and suggests hormone therapy or senolytics may mitigate these effects.

bioRxiv · Mar 2, 2023 · PreprintPathology

Dynamical modeling reveals RNA decay mediates the effect of matrix stiffness on aged muscle stem cell fate

Z. R. Hettinger, S. Hu, H. Mamiya, A. Sahu, H. Iijima, K. Wang, G. Gilmer, A. Miller, G. Nasello, A. D'Amore, D. A. Vorp, T. A. Rando, J. Xing, F. Ambrosio

This study shows that matrix stiffness affects aged muscle stem cell self-renewal through RNA decay dynamics, which can be modulated by young matrix environments.

bioRxiv · Feb 24, 2023 · PreprintCell Biology