H. E. Eaton

This study shows that breast cancer cells respond differently to low and high levels of progestins, with low levels promoting proliferation through specific molecular mechanisms.

This paper investigates how reovirus assembles its core and outer capsid proteins in distinct spatial and temporal compartments during replication, revealing a spatiotemporal segregation mechanism.

This study shows that breast tumor-associated metalloproteases cleave reovirus σ1 protein, reducing infectivity, and demonstrates that a σ1 mutation can overcome this inactivation.