Author

J. Gill

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:statistical model· 2 Research Focus:clustering· 1 Assay:CRISPR screen· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

A. Barka· 1 shared A. C. Wong· 1 shared A. Di Francesco· 1 shared A. Freund· 1 shared A. G. Deighan· 1 shared A. Raj· 1 shared A. S. Bhatt· 1 shared C. A. Thaiss· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperBiochemistry

A. Barka

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3 papers

Recent·Most cited

Interactions between the gut microbiome, dietary restriction, and aging in genetically diverse mice

L. Litichevskiy, M. Considine, J. Gill, V. Shandar, T. O. Cox, H. C. Descamps, K. M. Wright, K. R. Amses, L. Dohnalova, M. J. Liou, M. Tetlak, M. R. Galindo-Fiallos, A. C. Wong, P. Lundgren, J. Kim, , C. A. Thaiss

This study investigates how the gut microbiome interacts with aging and dietary restriction in mice, finding that host genetics significantly influence microbiome features and that dietary restriction does not rejuvenate the microbiome.

bioRxiv · Nov 28, 2023 · PreprintMicrobiology

Cooperativity between Cas9 and hyperactive AID establishes broad and diversifying mutational footprints in base editors

K. N. Berrios, A. Barka, J. Gill, J. C. Serrano, P. F. Bailer, J. B. Parker, N. H. Evitt, K. S. Gajula, J. Shi, R. M. Kohli

This study develops hyperactive base editors by exploring Cas9 and AID cooperation, revealing mechanisms that expand mutational diversity and efficiency in base editing.

bioRxiv · Dec 3, 2022Biochemistry

Expression Ratios of the Anti-Apoptotic BCL2 Family Members Dictate the Selective Addiction of KSHV-Transformed Primary Effusion Lymphoma Cell Lines to MCL1

D. Dunham, P. Viswanathan, J. Gill, M. Manzano

This study shows that PEL cells depend on MCL1 for survival due to its higher expression compared to other BCL2 family members, and suggests caution in using MCL1 inhibitors as monotherapy.

bioRxiv · Sep 7, 2022Microbiology