Author

J. Harding

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:scRNA-seq· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

A. Joseph· 2 shared D. A. Rauch· 2 shared L. Ratner· 2 shared A. Nagy· 1 shared C. Li· 1 shared D. Veis· 1 shared H. Yang· 1 shared J. Al-Saleem· 1 shared

Activity by Year

2019-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersMicrobiology

A. Joseph

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Role of the CTCF Binding Site in Human T-Cell Leukemia Virus-1 Pathogenesis

A. Joseph, X. Cheng, J. Harding, J. Al-Saleem, P. Green, D. Veis, D. A. Rauch, L. Ratner

This study shows that the CTCF binding site in HTLV-1 regulates viral pathogenesis by affecting proviral load, Tax expression, and T-cell activation in humanized mice.

bioRxiv · May 28, 2024Microbiology

Single-Cell Transcriptomic Analysis of Kaposi Sarcoma

D. A. Rauch, P. Valino Ramos, M. Khanfar, J. Harding, A. Joseph, O. Griffith, M. Griffith, L. Ratner

This study uses single-cell RNA sequencing to analyze Kaposi Sarcoma tumors and blood samples, identifying KSHV-infected cell types and their biomarkers, and showing changes with therapy.

bioRxiv · May 1, 2024Cancer Biology

Induction of long-term allogeneic cell acceptance and formation of immune privileged tissue in immunocompetent Hosts

J. Harding, K. Vintersten-Nagy, M. Shutova, H. Yang, J. Tang, M. Massumi, M. Izadifar, Z. Izadifar, P. Zhang, C. Li, A. Nagy

This paper demonstrates that expressing eight immunomodulatory genes in mouse embryonic stem cells enables them to avoid immune rejection and form immune-privileged tissues in allogeneic hosts.

bioRxiv · Jul 30, 2019 · PreprintBioengineering