Author

J. I. Warrick

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 4

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:metabolism & bioenergetics· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

D. J. DeGraff· 3 shared C.-W. Chen· 2 shared K. M. Aird· 2 shared V. Walter· 2 shared B. L. Worley· 1 shared D.-H. Shin· 1 shared E. S. Dahl· 1 shared G. B. Mills· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersCancer Biology

D. J. DeGraff

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4 papers

Recent·Most cited

Cancer BiologybioRxivPreprintMay 22, 2023

Urothelium-specific expression of mutationally activated Pik3ca initiates early lesions of non-invasive bladder cancer

L. Shuman, J. Pham, T. Wildermuth…·D. J. DeGraff

This study creates a mouse model to study PIK3CA mutations in bladder cancer, showing early tumorigenic changes but no progression to invasion without additional genetic factors.

DOI 10.1101/2023.05.22.541489

mouseproteomics

𝕏 PostBluesky

Cancer BiologybioRxivPreprintSep 11, 2020

Loss of the Pioneer Factor FOXA1 Results in Genome-wide Epigenetic Reprogramming and activation of Interferon-Response Genes including CD274/PD-L1

W. Hu, H. Yamashita, J. Craig…·D. J. DeGraff

This study shows that FOXA1 loss in bladder cancer causes widespread epigenetic changes and activates interferon-response genes like PD-L1.

DOI 10.1101/2020.09.11.294041

humanChIP-seq

𝕏 PostBluesky

Cancer BiologybioRxivJul 20, 2019

Suppression of p16 increases nucleotide synthesis via mTORC1

R. Buj, C.-W. Chen, E. S. Dahl…·K. M. Aird

This study shows that p16 suppression increases nucleotide synthesis through mTORC1 activation, promoting tumorigenesis by bypassing senescence.

DOI 10.1016/j.celrep.2019.07.084

humanexperimental assay

𝕏 PostBluesky

Cancer BiologybioRxivJun 13, 2019

Context-dependent activation of SIRT3 is necessary for anchorage-independent survival and metastasis of ovarian cancer cells.

Y. S. Kim, P. Gupta-Vallur, V. M. Jones…·N. Hempel

This study shows that SIRT3 activation is required for ovarian cancer cell survival and metastasis by regulating mitochondrial antioxidant defenses and metabolic changes during detachment.

DOI 10.1038/s41388-019-1097-7

humanproteomics

𝕏 PostBluesky