Author

J. Sutton

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 2 Cancer Biology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Assay:DNA methylation· 1

Frequent Co-Authors

K. Murphy· 2 shared A. Criscuolo· 1 shared A. De Reynies· 1 shared A. J. Williamson· 1 shared A. Lluch· 1 shared A. Lorincz· 1 shared A. Prat· 1 shared A. Sagrera· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersBiochemistry

K. Murphy

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Author profile →

5 papers

Recent·Most cited

Comprehensive Characterization of tRNA by Ultra High-Performance Liquid Chromatography High-Resolution Accurate Mass Spectrometry

R. L. Ross, R. Cowley, K. Murphy, R. Gamez, P. Gazis, J. Sutton, M. Du

The paper characterizes tRNA modifications using high-resolution mass spectrometry and identifies novel features in yeast tRNA.

bioRxiv · Jun 7, 2023 · PreprintBiochemistry

Characterisation and sequence mapping of large RNA and mRNA therapeutics using mass spectrometry

C. Vanhinsbergh, A. Criscuolo, J. Sutton, K. Murphy, A. J. Williamson, K. Cook, M. J. Dickman

This study develops an automated workflow for rapid characterization and sequence mapping of large RNA and mRNA therapeutics using mass spectrometry.

bioRxiv · Feb 14, 2022Biochemistry

Colorectal cancer risk in bowel adenomas based on lifestyle exposures, tissue preconditioning and DNA methylation

J. Sutton, M. Moorghen, L. M. Wang, C. Thirlwell, C. Pipinikas, A. Lorincz

This study identifies DNA methylation changes linked to high alcohol and BMI exposures in colorectal adenomas, suggesting they may increase CRC risk through tissue preconditioning.

bioRxiv · Aug 26, 2021 · PreprintCancer Biology

The GATA3 X308_Splice breast cancer mutation is a hormone context-dependent oncogenic driver

N. Hruschka, M. Subijana, O. Grana-Castro, F. Del Cano-Ochoa, L. Pare Brunet, A. Sagrera, A. De Reynies, D. Andreu, J. Sutton, I. Chernukhin, S.-F. Chin, C. Caldas, A. Lluch, O. Burgues, B. Bermejo, , P. Martinelli

The paper investigates the context-dependent oncogenic effects of the GATA3 X308_Splice mutation in breast cancer, showing its differential impact based on hormonal status.

bioRxiv · Jun 8, 2019Cancer Biology

Streptococcus agalactiae induces placental macrophages to release extracellular traps loaded with tissue remodeling enzymes via an oxidative-burst-dependent mechanism

R. S. Doster, J. Sutton, L. M. Rogers, D. M. Aronoff, J. Gaddy

This paper shows that placental macrophages release extracellular traps containing enzymes that kill GBS and may contribute to fetal membrane damage during infection.

bioRxiv · Oct 11, 2018Microbiology