Author

J. Talukdar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

B. Das· 2 shared B. Pal· 2 shared L. Pathak· 2 shared S. Bhuyan· 2 shared S. Gayan· 2 shared S. Sandhya· 2 shared A. Alshamsan· 1 shared A. Malik· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersImmunology

B. Das

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Overexpression of Interleukin-8 and Interleukin-13 as potent immune markers associated with survival and dietary habits in esophageal squamous cell carcinoma.

J. Talukdar, A. Malik, K. Kataki, B. N. Choudhury, M. N. Baruah, M. Bhattacharyya, M. Alkholief, A. Alshamsan, M. P. Sarma, M. Bhattacharjee, M. Basak, M. P. Kashyap, S. Bhattacharjee, E. Ali, C. Keppen, , S. Medhi

The paper evaluates the expression of IL8, IL12, and IL13 in ESCC patients and links their levels to survival, clinicopathological factors, and dietary habits.

bioRxiv · Oct 17, 2022 · PreprintImmunology

Stem cell basis of a host driven transmission of antigen packed aerosols: a novel mechanism of natural vaccination for tuberculosis

B. Das, L. Pathak, S. Gayan, B. Pal, P. j. Saikia, T. Baishya, N. R. Das, R. Das, M. Maral, R. Mahanta, S. Bhuyan, P. Gautam, J. Talukdar, S. Sandhya, D. Kalita, , I. Pulu

The paper investigates a novel natural vaccination mechanism where tuberculosis-infected individuals spread antigen-loaded extracellular vesicles via aerosols, inducing herd immunity in healthy mice.

bioRxiv · Nov 14, 2020 · PreprintImmunology

Coronavirus activates a stem cell-mediated defense mechanism that reactivates dormant tuberculosis: implications in COVID-19 pandemic

L. Pathak, S. Gayan, B. Pal, J. Talukdar, S. Bhuyan, S. Sandhya, H. Yeger, D. Baishya, B. Das

The paper shows that coronavirus infection in mice reduces viral loads by reactivating dormant tuberculosis through stem cell reprogramming, suggesting a potential defense mechanism with implications for COVID-19 vaccine development.

bioRxiv · May 6, 2020 · PreprintMicrobiology